For as long as we've known that soil bacteria manufacture molecular weapons to fight each other, we've been swiping their battle plans. In clinics and hospitals, those turf-war weapons have become miraculous drugs of modern medicine—antibiotics—that blow away otherwise deadly infections.
But, of course, there's a dark side of mimicking microbial munitions—bacteria have defenses, too, namely antibiotic resistance. You're probably aware that we're facing a rising threat of drug resistance among disease-causing bacteria, one that is rendering much of our stolen weaponry obsolete and making infections harder to defeat.
Often, this growing crisis is framed as a clinical failure: We're overusing and misusing antibiotics, hastening our bacterial foes' natural ability to develop and spread resistance. While this is certainly true, a new study in Nature Microbiology this week identifies a potentially new driver of rising antibiotic resistance—and we're at least partly to blame for this one, too.